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Cigarette smoke extract profoundly suppresses TNFα-mediated proinflammatory gene expression through upregulation of ATF3 in human coronary artery endothelial cells

Endothelial dysfunction caused by the combined action of disturbed flow, inflammatory mediators and oxidants derived from cigarette smoke is known to promote coronary atherosclerosis and increase the likelihood of myocardial infarctions and strokes. Conversely, laminar flow protects against endothel...

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Main Authors: Teasdale, J.E., Hazell, G.G.J., Peachey, A.M.G., Sala-Newby, G.B., Hindmarch, C.C.T., McKay, T.R., Bond, M., Newby, A.C., White, S.J.
格式: Article
語言:English
出版: Nature Publishing Group 2017
主題:
R Medicine
在線閱讀:http://eprints.um.edu.my/19006/1/Cigarette_smoke_extract_profoundly_suppresses_TNF%CE%B1-mediated_proinflammatory_gene_expression_through_upregulation_of_ATF3_in_human_coronary_artery_endothelial_cells.pdf
http://eprints.um.edu.my/19006/
http://dx.doi.org/10.1038/srep39945
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http://eprints.um.edu.my/19006/1/Cigarette_smoke_extract_profoundly_suppresses_TNF%CE%B1-mediated_proinflammatory_gene_expression_through_upregulation_of_ATF3_in_human_coronary_artery_endothelial_cells.pdf
http://eprints.um.edu.my/19006/
http://dx.doi.org/10.1038/srep39945

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